Fibromyalgia often consists of moving pains that alternate joints. There are two different factors often involved. One is mediated by nerves and the other by local issues to the joints.

Nerve Fiber Cause

In the brain immediately behind the limbic area, which is relatively central in the cerebrum, there are fibers that connect the pineal gland to the midbrain that also cross through the amygdala. These fibers are called the “Pineal Midbrain Fibers” within EM. These fibers are unique in the body in that they appear to be extremely sensitive to any kind of stimulation. Any caustic stimulus to these fibers causes pain to be felt somewhere in the body often far away in the arms and legs. While they are incredibly well protected from physical trauma, they unfortunately are still capable of being damaged by pathogens and their pathogen toxins.

EM Wounds and/or fissures at these fibers are common causes of fibromyalgia pain. A common cause of wounding and fissures is the pathogens in EM we associate with Streptococcus. Streptococcus Thermophilus commonly infects the sinus chamber between the front and back sinuses. Eventually, the acid it produces burns holes through the skull bone, the meninges, and into the brain and the pathogen then deepens its infection into the brain itself. Histio of hydrochloric acid is the most common causative factor of this form of fibromyalgia. To successfully treat this issue, you need to clear this histio, attack the strep pathogen, and treat with the EM Wounds Treatment and the EM Fissures Treatment. Another possible problem that can arise is separation of the synaptic junctions in the pineal gland, pineal midbrain fibers, and/or midbrain. To treat this use the EM Neurexin And Neuroligin Array Treatment which tightens the neurexin and neuroligin structures that brace the synaptic junction and hold it together.

Another possibility is infection of the pineal gland from a pathogen that generates swelling and edema. A common pathogen that can get into this location is one we associate with propionibacterium Acne which generates hot hormone arrays, progesterone histio, swelling, Fire Qi excess in the blood vessels, edema, cortisol histio, and EM Tighten Tissues. As a result, it makes the pineal gland bulge and force the pineal midbrain fibers to bend and buckle. This kind of stimulus is very bad for these fibers and will cause severe pain. See the Protocols in the beginning of this curriculum for ideas as to how to clear the swelling and edema. Also attack propionibacterium acne associated pathogens.

It is very important to understand that any stimulus is bad for these fibers – including the stimulus necessary to rectify the problem. It is very common that pain increases for several days after repairing the imbalances to the pineal midbrain fibers.

Local Causes

Another possible cause of fibromyalgia relates to local imbalances at the afflicted joints. Pathogens impacting the blood vessels at these joints can lead to pain upon flexion and extension of the joint – not because of any soft tissue or nerve issues but because the blood vessels also must bend at the joint and they are too swollen or edemous to bend painlessly.

The most common cause of this form of fibromyalgia is damp pathogens in the form of fungus in the blood vessels. Fungus prefers to grow in the “dark spaces” of the body just like it prefers that in the environment. It prefers to grow in bones but in order to find its way to bones it often grows colonies in veins. Fungus associated pathogens produce hot hormones which over dilate the blood vessels leading to edema. Edema in a blood vessel is not like edema in an interstitial space. There is a limit as to how far it can grow before pressures build inside the vein making the vein taut and far more sensitive to movement. In order to treat this form of fibromyalgia it is necessary to attack fungus and especially the EM pathogen we associate with Aspirgillis. It is also important to follow the Edema Protocol to clear the edema so that the vein can return to proper shape and normal flexibility.